What is the role of beta blocker in patients with heart failure?

What is the role of beta blocker in patients with heart failure?

What is the role of beta blocker in patients with heart failure?

Beta‐blockers may also reduce the risk of arrhythmia, improve LVEF, improve symptoms of heart failure, and may control ventricular rate (Chatterjee 2013; Dargie 2001).

What happens if a healthy person takes a beta blocker?

Side effects commonly reported by people taking beta blockers include: feeling tired, dizzy or lightheaded (these can be signs of a slow heart rate) cold fingers or toes (beta blockers may affect the blood supply to your hands and feet) difficulties sleeping or nightmares.

Do you give beta blockers for heart failure?

If you have heart failure, you need beta-blockers — even if you do not have symptoms. Beta-blockers are prescribed for patients with systolic heart failure and improve survival, even in people with severe symptoms.

Which beta blocker have mortality in heart failure patients?

Summary of Published Mortality Trials of Metoprolol in Patients with Heart Failure. Treatment improved clinical status, reduced the need for heart transplantation and was safe. Study was stopped early. Treatment reduced sudden deaths and deaths from progressive pump failure.

Which beta blocker is best in heart failure?

Apart from the two second generation beta blockers metoprolol and bisoprolol, carvedilol has also been shown to be effective in heart failure to reduce morbidity and mortality.

Can beta-blockers worsen heart failure?

However, beta blockers may exacerbate symptoms in patients with acute decompensated heart failure or in those with preexisting myocardial dysfunction and borderline compensation, since the maintenance of cardiac output in such patients depends in part upon sympathetic drive.

Do beta-blockers improve mortality in heart failure?

28 This analysis revealed that beta blockers reduced all-cause mortality by 32 percent (P = 0.003), reduced the combined risk of death or hospitalization because of heart failure by 37 percent (P < 0.001) and increased the ejection fraction by 29 percent (P < 10−9).